Research Findings Show COVID-19 mRNA Vaccines Alter Genes and Cause Permanent Heart Damage

COVID-19 vaccines are, in reality, gene-altering therapies that have long-term deleterious effects on the health of the recipients, myocarditis being one of the side effects.

May 11, 2025

A research study published in the International Journal of Cardiovascular Research & Innovation presents evidence that proves beyond a reasonable doubt that the COVID-19 mRNA vaccines cause debilitating myocarditis. The study concludes that the COVID-19 vaccines are, in reality, gene-altering therapies that have long-term deleterious effects on the health of the recipients, myocarditis being only one of the side effects.

COVID-19 mRNA products are, by definition, gene therapy products that rely on synthetic, modifed mRNA (or adenoviral vector DNA), encapsulated within a protective lipid nanoparticle vehicle. These nucleic acids are designed to instruct the ribosomes in the body’s cells to produce the SARS-CoV-2 spike protein, the intended target antigen. Although the Pfizer and Moderna products are different, they both utilize the synthetic, modifed mRNA, which encodes for the same recombinant spike protein corresponding with the protein identifed on the surface of SARS-CoV-2. For this reason, both mRNA products could also be labeled “gene therapy vaccines.”

Id. at page 3.

The researchers uncovered a trick used by governments and pharmaceutical companies to conceal the heart injuries caused by the COVID-19 vaccines by mischaracterizing the myocarditis as being caused by the alleged SARS-Cov-2 virus instead of the COVID-19 mRNA vaccines.

Because of the temporal ambiguity in COVID-19, it is logical to postulate that the misdiagnosis of myocarditis among athletes would be more likely to result in a miscategorization of “COVID-related myocarditis”, thus reinforcing the false claim that these cases are more commonly linked with the coronavirus infections than with the mRNA vaccines. … Ultimately, while mRNA vaccine-related myocarditis cases show a clear temporal association with the vaccines, infection-related cases are subject to both denominator inaccuracy and a prolonged window of potential attribution, with elevated rates of incidental COVID-19 hospitalizations.

Id. at page 16.

The researchers revealed how statistics and studies were massaged to conceal the injuries caused by the COVID-19 vaccines.

Flawed study designs, methodologies, and reporting practices, all of which may greatly compromise the integrity of the data and reliability of the findings. For example, as we noted previously, most observational studies have used the CDC definition of “vaccination status”: each individual is considered “unvaccinated” until 14 days after dose 2. Conversely, individuals are counted as “vaccinated” only 14 days after the second dose of a two-dose COVID-19 vaccine series. This practice has two fundamental implications: (1) individuals testing positive for SARS-CoV-2 before this period, any time prior to the 14 days after dose 2, are considered “unvaccinated”, or not sufficiently protected; and (2) any AE [adverse event] occurring after the first dose or within 14 days of the second dose will be classified as occurring among the “unvaccinated” and counted as such.

Id. at page 17.

That statistical flim-flam had the effect of concealing myocarditis vaccine injuries.

[A]ll myocarditis cases occurring in the rst 3-4 weeks of the first injection, or in the first 2 weeks following the second injection, will be classified as “unvaccinated”. This outcome has resulted in the common yet erroneous belief that the mRNA vaccines prevent more myocarditis than they cause, and therefore mRNA-attributable myocarditis is rare. This problem was intrinsic to the many studies that adhered to the CDC definition of vaccination status.

Id. at page 17.

Thus, any cardiac events caused by the mRNA COVID-19 vaccines that happened shortly after the injection were classified as cardiac events among the “unvaccinated” population. The cardiac event was then falsely attributed to the alleged SARS-CoV-2 virus rather than the mRNA COVID-19 vaccine.

In epidemiological terms, because of the CDC’s “non-vaccinated” 2-week rule, any cardiac events occurring within the specified timeframe are misclassified as events among the “unvaccinated”, leading to extremely biased estimates of risk associated with the mRNA products. This also overestimates myocarditis cases attributed to SARS-CoV-2 infection because, again, any test-positive individual who develops myocarditis prior to 14 days after their second mRNA dose are classified as “unvaccinated”, with a diagnosis of “infection-related myocarditis”. This misclassification skews the data, as early mRNA vaccine-related myocarditis cases are excluded from the “vaccinated” cohort and reassigned to the “unvaccinated” or infection-related cohort. This explains how, in many large observational studies, myocarditis cases caused by the mRNA injection are easily misattributed to SARS-CoV-2 infection. Whereas the risk of infection-related myocarditis is inflated, the risk associated with mRNA products is profoundly underestimated.

Id. at page 17.

Governments and the scientific community try to downplay the seriousness of myocarditis caused by the COVID-19 vaccines by characterizing the resulting heart damage as “mild myocarditis.” So-called mild myocarditis leaves permanent scarring of the heart muscle that can have life-long consequences.

Even with mild symptoms, the underlying physiological effects and anatomical scarring may be more severe or potentially lethal in the long term. Myocarditis may eventually develop into congestive heart failure, dilated cardiomyopathy, cardiogenic shock (severe reduction in cardiac output leading to systemic hypoperfusion and organ failure), and, in some instances, sudden cardiac death.

Id. at page 20.

The reason that it is a misnomer to characterize myocarditis as “mild” is that damage to the heart muscle is usually permanent. Unlike skeletal muscles, the heart muscle cannot self-repair through regeneration. Instead, the damaged heart muscle is replaced by permanent scar tissue.

Unlike skeletal muscle, cardiac muscle displays a limited capacity for regeneration following injury. Instead of regenerating functional myocytes, damaged cardiac tissue is replaced by brotic scar tissue, which is permanent. This structural remodeling disrupts the electrical and mechanical integrity of the myocardium, increasing the risk of arrhythmias and contributing to a higher likelihood of premature death over the individual’s lifetime.

Id. at pages 20-21.

Myocarditis is always serious because the damage to the heart is permanent. A person may have asymptomatic myocarditis but can nonetheless have a lifelong risk of sudden congestive heart failure and premature death. Up until their death, they would have no clinical signs of heart trouble. They would be living their (shortened life) with a vaccine-damaged heart that without warning suddenly gives out. The evidence of the heart damage would be discovered only after the person died. In a discussion this author had with the Associate Press (AP) on or about November 16, 2022, about vaccine-induced myocarditis, I was told by the AP that experts consulted by the AP opined that “most myocarditis cases are indeed mild or asymptomatic.” But the AP experts’ opinion raises an issue. How can a doctor know to look for myocarditis if it is asymptomatic? It would seem that the only time that asymptomatic myocarditis would be detected is after the person suddenly and unexpectedly dies or is hospitalized. Otherwise, if the patient is asymptomatic there is no complaint for the doctor to examine the patient for myocarditis. It seems that asymptomatic myocarditis is a very serious and perilous condition indeed. And that is precisely the conclusion of the study. Asymptomatic myocarditis can be deadly.

Emergent evidence suggests the possibility of myocardial fibrosis and potential long-term sequelae in symptomatically mild or even asymptomatic or subclinical myocarditis. These “mild clinical cases” can involve severe cardiac fibrosis (scarring), with permanent damage to the heart muscle and a lifelong risk of potentially fatal arrhythmias. Over time, such damage can progress to congestive heart failure and premature death. The pivotal Pfizer and Moderna trials were not designed to capture these long-term risks, most of which only became apparent after 2.5 years of follow-up and the administration of over a billion mRNA doses.

Id. at page 21.

The asymptomatic heart damage seems to be endemic among athletes, who, without warning, drop dead suddenly from heart failure due to a vaccine-damaged heart.

The myocardial damage associated with these vaccines seems to result in sudden death due to the added stress of intensive physical exertion (or exercise intolerance) as a byproduct of the elevated catecholamine levels commonly seen in male athletes and further exacerbated by the mRNA product’s components.

Id.

One of the ways that the government agencies concealed the harm caused by the COVID-19 vaccines was to claim the damage was caused not by the vaccine but by the SARS-CoV-2 virus. They ramped up the alleged myocarditis cases in the unvaccinated population by mischaracterizing vaccinated persons as being unvaccinated and by associating the heart damage caused by Remdesivir (brand name, Veklury), an unsafe and ineffective drug used to treat COVID-19, as myocarditis caused by SARS-CoV-2.

One factor that may have contributed to the misperception that COVID-related myocarditis tends to be more severe than mRNA vaccine-related myocarditis is the drug known as Remdesivir (brand name, Veklury).

Id. at page 23.

Remdesivir became the primary COVID-19 drug approved for use in U.S. hospitals, with the U.S. government paying hospitals a 20% bonus incentive for utilizing the Remdesivir protocol.
Eventually, however, a controlled clinical trial showed that Remdesivir failed to provide any significant clinical benefit. Part of this failure was due to the drug’s poor safety profile and its ability to induce persistent mitochondrial and structural damage in human cardiomyocytes. Clinically, Remdesivir can have significant cardiotoxic effects, potentially leading to prolonged QT intervals and torsade de pointes, which in turn can result in ventricular arrhythmias and sudden cardiac arrest. The drug was also linked with sinus bradycardia (slow heart rate), which can cause serious complications, notably heart failure and cardiac arrest. Remdesivir’s cardiotoxic impact may be heightened in patients with pre-existing cardiovascular conditions.

Id. at page 23-24.

Clinical autopsies have detected mRNA-derived spike proteins in the damaged hearts of decedents, thus indicating the cause of the heart damage.

In terms of biological plausibility for the more serious cardiac sequelae, both the synthetic, modified mRNA and mRNA-derived spike protein have been detected in the hearts of individuals who died following the COVID-19 mRNA vaccines and in cases of mRNA vaccine-related myocarditis, respectively. In contrast, autopsy findings from individuals who died following SARS-CoV-2 infection suggest that any incidental myocarditis is not associated with direct cardiac infection with the coronavirus.

Id. at page 24.

The researchers concluded that all mRNA COVID-19 vaccines be given a black-box warning highlighting “myocarditis, pericarditis, and myocarditis as potentially life-threatening adverse effects.”

In conclusion, rare viral myocarditis, pericarditis, and myocarditis can often present with mild clinical symptomology. Acute vaccine-induced myocarditis, presents with acute symptoms, arrhythmias, and heart failure warranting hospitalization. Despite hospitalization, vaccine myocarditis has led to death. Even minimal inflammation or scarring of the cardiac musculature—often indicated by elevated cardiac troponin levels—can predispose young individuals to a lifelong increased risk of heart failure and sudden cardiac arrest. Even cases classified as mild myocarditis may result in persistent cardiovascular complications, including chronic cardiac dysfunction and arrhythmias. Given these potential outcomes, the COVID-19 mRNA vaccine products warrant a black box warning to highlight myocarditis, pericarditis, and myocarditis as potentially life-threatening adverse effects.

Id. at page 24.